The link between Hashimoto’s and the Herpes rash is this. If you have any thyroid issue whether it is Hashimoto’s, low thyroid or hyperthyroid. You will have an issue with gluten sensitivity.
Is Your Herpes Break Out Really a Gluten Rash?
Are you suffering from occasional herpes out breaks on areas of your body but not on the genitals? Those with a sensitivity to gluten, often develop blistering rashes that are often mistaken for a herpes outbreak.
A Local Dermatologist told us he is upsetting the GI docs because he is diagnosing gluten sensitivity/celiac more than they are. He is sending patients out to be tested for gluten sensitivity/celiac when he sees these blistering rashes. He says if the fluid from one of these blisters is collected and tested it comes back very frequently positive for gluten sensitivity. However, most these rashes are so itchy, patients scratch and break the blisters before it can be collected.
Call today! 530-615-4083
Are you frustrated because the herpes medication doesn’t seem to control it or provide any relief? A good way to control the outbreaks would be to identify the causes, and take effective measures to ensure that they remain out of your daily life. How was your herpes diagnosed? What if your herpes outbreak wasn’t caused by virus. What then?
Dermatitis herpetiformis (DH), an itchy, stinging, blistering skin rash, occurs when your skin reacts to gluten antibodies circulating in your system. Some people call dermatitis herpetiformis a “gluten rash” or a “celiac disease rash” because it occurs in conjunction with a sensitivity to gluten. Regardless of the presentation or symptoms, a positive diagnosis of DH always indicates that a gluten sensitive enteropathy is occurring.
The current dogma is that Immune cells are stimulated by Antibodies. They believe as long as antibodies are present, the antibodies are constantly stimulating an immune response. They also are lead to believe the presence of antibodies are from an ongoing infection. Typically, IgG antibodies are produced in the first phase of immune responses. However, the immune response to IgA, IgG, and IgM declines or is terminated along the course of the disease in most patients. IgM antibodies are indicative of an ongoing immune response., Studies have shown the detection of IgA and IgM antibodies in 62% and 61% of IgG antibodies in autoimmune patients, and after 7 years.,
A resolution of the disease is not associated with a decrease in antibodies. Only those with a short disease duration are likely to experience a decrease in antibodies levels. For most autoimmune patients there is no significant change in antibody levels, However, patients with immune calming/quieting treatment, experienced a 20% decline in their antibody levels. Normalization in the levels of antibodies with treatment intervention occurs in less that 7% of patients. The success of clinical interventions in lowering antibody levels (and thus likely minimizing the pathogenic effects of autoantibody binding) appears to be dependent on disease duration. The association of shorter disease duration with greater declines in antibody levels is highly consistent with the growing body of evidence that shows improved clinical outcomes with earlier disease intervention in autoimmunity.,,
Approximately one-half of patients with Rheumatoid Arthritis had positive IgM-RF and/or antibodies on at least 1 occasion, almost 5 years prior to disease onset. More than 50% of autoimmune patients may have positive antibodies upwards of 13 years after disease resolution, regardless of treatment.
Dermatitis herpetiformis (DH) can often be misdiagnosed and frequently confused with skin conditions such as: allergies, bug or mosquito bites, contact dermatitis, diabetic pruritus, eczema, herpes, shingles, hives and psoriasis.
Reddened skin, circular bumps, and blisters filled with clear, neutrophil (white blood cell), containing liquid are very common. Skin lesions and scarring can also appear, particularly in patients that scratch and irritate the skin during outbreaks.
The onset of DH may be acute or gradual, appearing in the same location every time. DH outbreaks are very often mirrored, meaning that the rash will occur on both sides of the body in exactly the same place. The hallmark sign of DH is an intensely itchy, blistering skin rash. For people that seem to suffer more often than not with these outbreaks, trying to use cold sore medicine variations could potentially decrease the itchiness and halt the outbreak before it gets any worse.
DH can appear anywhere on the body; however, It most frequently present in the following areas:
- Lower back
Dermatitis herpetiformis sufferers usually experience to rash in the same location every time. The rash might be continuous, or it might come and go.
Before the actual dermatitis herpetiformis breaks out, your skin may itch in that location, or it might feel is if it’s burning. The rash itself usually includes reddened skin plus multiple small, pimple like-bumps which contain a clear liquid.
The dermatitis herpetiformis bumps usually take several days to heal (during which time new bumps usually appear nearby), and once healed, only behind small purple marks that lasts for weeks or months. People with long-standing dermatitis herpetiformis usually have continuously reddened skin where the rash occurs.
As the name implies, dermatitis herpetiformis (DH) looks herpetic and is sometimes mistaken for a herpes virus. Red plaques erupt in groups of blisters, in the resulting intense itch can disturb sleep.
Who does dermatitis herpetiformis affect?
Unlike gluten sensitive enteropathy or celiac disease, which is diagnosed more often in women, dermatitis herpetiformis is more common in men. In fact, some studies show a male-to-female ratio of up to 2-to-1 in dermatitis herpetiformis patients. Men are more likely to have a typical oral or genital lesion.
DH can affect people of all ages but most often appears for the first time between the ages of 15 and 40. People of northern European descent are more likely than those of African or Asian heritage to develop DH.
Dermatitis Herpetiformis is an autoimmune blistering disorder associated with a gluten sensitive enteropathy. People with DH have a condition of the intestinal tract identical to that found in gluten sensitive enteropathy or celiac disease, although gastric symptoms might be absent. Autoimmune processes can be arrested if the interplay between the Neuro-Endo-Immune Supersystem and environmental triggers is prevented by re-establishing intestinal barrier function.
How does a disorder that damages the intestines show up on the skin?
When a person has gluten sensitive enteropathy consumes gluten, the mucosal immune system in the intestines responds by producing a type of antibody called Immunoglobulin A. As IgAs enters the bloodstream, they can collect in small vessels under the skin, triggering further immune reactions that result in the blistering rash of DH.
The first clue that a skin eruption may be DH is that “it itches like crazy.” People are digging at themselves. As a result the blisters are almost always broken open by the time the DH suffer seeks medical help.
The second characteristic sign of DH is its location on the body. Lesions most often appear on the extensor surfaces – the forearms near the elbows, the knees, and the buttocks. The outbreak of the lesions also tends to be bilateral, meaning it appears on both sides of the body.
The grouping of the lesions provides a final clue. Although DH is not caused by a herpes virus, its lesions resemble those of herpes and hence the word herpetiformis. In both conditions, lesions are formed in small groups. Still, DH is often confused with eczema, a common inflammatory skin disorder that, like DH, results in an itchy rash that is often scratched raw.
A gluten sensitive enteropathy is different from celiac disease in that a gluten sensitivity is the result of a microscopic colitis. The damage to the gastrointestinal lining in microscopic colitis is not bad enough for a biopsy or endoscopy to see enough damage to be called celiac disease. Many patients report they have been checked by their doctor and were told their lining was red or irritated. So they did not have a gluten sensitivity because they did not have celiac. Many healthcare providers also assume a person’s immune system is always working at 100% optimal efficiency. When in fact, the immune system is so fatigued from constant exposure to gluten. It cannot create a response in the normal fashion.
Breaking with Bread
A strict gluten – free diet may take weeks to clear up an outbreak. Accidental ingestion of gluten will cause symptoms to recur. Hidden sources of gluten can prolong the outbreak.
For some it will be as easy as maintaining a gluten free lifestyle. Others will require our help in quenching the immunoglobulin reaction, controlling the immune response, restoring the digestive chemistry and repairing the gastrointestinal lining to enable them to maintain themselves through diet and lifestyle.
Call today! 530-615-4083